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Repage (Repaglinide)
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Repaglinide

Both Repage and gliclazide are insulin secretagogues widely used in the treatment of type 2 diabetes. The insulinotropic action of the meglitinide analogues KAD-1229, A-4166 and Repage was examined in rat pancreatic islets deprived of exogenous nutrient or incubated in the presence of nutrient secretagogues such as D-glucose and the methyl esters of pyruvic acid, succinic acid and glutamic acid.

Other names for this medication:
Repaglinide, Prandin, Glimet, Novonorm, Premil, Regan, Repaglinida, Eurepa, Repage, Page, Repa,

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Repage is used together with diet and exercise to treat high blood sugar (glucose) levels in patients with type 2 diabetes. Repage works by causing your pancreas to release more insulin into the blood stream. This medicine does not help patients who have insulin-dependent or type 1 diabetes.

This medicine is available only with your doctor's prescription.

This product is available in the following dosage forms:

  • Tablet

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Reduction of mastitis caused by experimental challenge with Staphylococcus aureus and Streptococcus agalactiae by use of a quaternary ammonium and halogen-mixture teat dip. C substance and M protein of Group A streptococci released from the cell by sonic oscillation were toxic to the adrenalectomized rat in quantities of 1 mg./100 gm. Finally, a neutralizing antibody against one bound protein was added to the cell cultures to repeat the osteogenic assays to clarify its role in the in vitro effects of the P-15/anorganic bovine mineral combination. A systematic review of the experiences and perceptions of the newly qualified nurse in the United Kingdom. The aim of this paper is to describe the development of a complex intervention of peer support in type 2 diabetes for a randomised control trial in a primary care setting.

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Towards a reconceptualization of striatal interactions between glutamatergic and dopaminergic neurotransmission and their contribution to the production of movements. A fourth patient underwent an exploratory laparotomy on the basis of clinical and CT scan findings that could have been circumvented with preoperative ERP. Efficacy was defined as the cessation of seizures after administration of ivLCM, with no need for any further antiepileptic drug.

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23 Tea Leaves II, has been developed and characterized by the National Institute for Environmental Studies (NIES), Japan. Fitting of conduction velocities provided a racemic bupivacaine to levobupivacaine and to ropivacaine ratio of 1:1.38 for concentration effect at 1,000-ms pacing cycle length, and 1:0.74 for use-dependent effect at 600-ms pacing cycle length. They represent lesions/conglomerates of abnormally configured vessels leading to seizures either as a result of physiological changes affecting the cerebral cortex immediately surrounding the CCM (an epileptogenic mechanism that is relevant for both temporal and extratemporal lesions), or as a result of promoting epileptogenicity in remote but anatomo-functionally connected brain regions (a mechanism that is particularly relevant for temporal lobe lesions).

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These models represent strains of MTBC using high-throughput biomarkers called spacer oligonucleotide types (spoligotypes), since these are routinely gathered from MTBC isolates of tuberculosis (TB) patients. Participation of the Golgi apparatus of certain root cells in the absorption of metallic salts lactis, homologous to a Bacillus stearothermophilus Tpi, is capable of enhancing bacteriocin synthesis in Lact. These unique contributions of the discipline of biomedical informatics are reviewed here.

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